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The results of measurements of T98G glioblastoma cells showed the opposite effect on cell viability
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Moreover, the kinase portion of IRE1 is capable of activating the adaptive tumor protein tumor necrosis α (Tnf- α ) receptor-associated factor 2 (TRAF2) and, subsequently, the NF κ B pathways and c-Jun N-terminal kinase (JNK) pathway [83]
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